When GERD Causes Persistent Cough

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There are several ways to significantly reduce the occurrence of acid reflux symptoms, and in most cases prevent the acid reflux before it starts. With less acid reflux episodes, there is less chance of esophageal damage. When stomach acid backs up (known as acid reflux) into the esophagus and is breathed in, it can cause coughing. Irritation from the acid reflux in the throat can also cause coughing.

What do we see in terms of mucosal damage? An otolaryngologist would describe a variety of classic findings from laryngopharyngeal reflux.

Acid reflux creates a burning pain in the lower chest area, often after eating. Exact figures vary, but diseases resulting from acid reflux are the most common gut complaint seen by hospital departments in the United States.

Gastroesophageal reflux disease (GERD) is the main etiologic factor of erosive esophagitis (“reflux esophagitis”). The prevalence of esophagitis is less than 10% in the general population, and approximately 30% in patients with gastroesophageal reflux symptoms.

Patients with this disease tend to be younger than those with endoscopy-positive disease and are more likely to be female, thin and without hiatal hernia. The trials have shown that, in patients with endoscopy-negative reflux disease, symptoms (particularly heartburn) tend to be less severe and less frequent than in patients with endoscopy-positive disease. The overlap between the groups is such, however, that the symptoms cannot be used to distinguish reflux patients with oesophagitis from those without. Approximately 50% of patients with endoscopy-negative disease were found to have levels of oesophageal acid exposure that fell within the normal range. Oesophageal pH monitoring is therefore of limited value in patients with endoscopy-negative reflux disease, unless the test focuses on analysis of the correlation between symptoms and episodes of reflux.

The other 33% of patients required an increase in medication to lower acid output to less than 5 mEq/h in 7% and less than 1 mEq/h in the other 26% to resolve symptoms and signs completely. We conclude that reflux esophagitis occurs in the majority of patients with Zollinger-Ellison syndrome and responds well to medical therapy, although one third of patients require intensive antisecretory medication. Gastroesophageal reflux disease (GERD) is the long-term, regular occurrence of acid reflux.

Symptoms of erosive esophagitis are not different from symptomatic gastroesophageal reflux, i.e., mainly heartburn, acid regurgitation, and chest pain. Dysphagia may be present whatever the severity of endoscopic lesions, and even in the absence of esophageal stricture. Proton pump inhibitors are the mainstay of medical therapy of reflux esophagitis, providing high healing rates (90%) and symptom relief (70-80%).

  • When that didn’t help, he moved it up to 80 milligrams a day, but I still had the chest pains.
  • Furthermore, we don’t have a validated instrument to define GERD in patients with laryngopharyngeal reflux.
  • The state of the art of GERD is described, based on recent definitions, pathophysiological evidence, epidemiology in developed countries, clinical subtypes together with a diagnostic approach specifically focussed on the appropriateness of endoscopy.
  • Patients with this disease tend to be younger than those with endoscopy-positive disease and are more likely to be female, thin and without hiatal hernia.
  • However, few studies have discussed on its relationship with metabolic syndrome (MetS).

How to Increase Stomach Acid at Home

Furthermore, we don’t have a validated instrument to define GERD in patients with laryngopharyngeal reflux. A variety of findings in the larynx can be nonspecific, such as erythema, edema, swelling, and cobblestoning. These findings can be induced by other conditions, such as postnasal drip, allergies, asthma, voice abuse, and even by repetitive behaviors such as throat clearing.

The state of the art of GERD is described, based on recent definitions, pathophysiological evidence, epidemiology in developed countries, clinical subtypes together with a diagnostic approach specifically focussed on the appropriateness of endoscopy. Metabolic syndrome is accurately defined and the pivotal role of insulin resistance is emphasized. The strong relationship between GERD and metabolic syndrome has been pathophysiologically analyzed, taking into account the role of obesity, mechanical factors and metabolic changes. Data collected by our group regarding eating habits and GERD are briefly summarized at the end of a pathophysiological analysis. The literature on the subject strongly supports the possibility that lifestyle and eating habits may be involved in both GERD and metabolic syndrome in developed countries.

Patients with MetS have a high risk of cardiovascular disease, diabetes mellitus, and other atherosclerotic diseases [16,23,24]. A retrospective case-control study in China in 2010 found that a high waist-hip ratio, hyperglycemia, hypertriglyceridemia, and MetS were the associated factors for RE, and that HDL-C was associated with a reduced risk of RE in men [25]. This population- based cross-sectional study was conducted in a one-stage randomized clustered sample of adult inhabitants in Kerman city in 2011-2012. A total of 2265 subjects with age range of 15-85 years were enrolled.

Gastroesophageal reflux disease (GERD) is one of the most common gastrointestinal problems worldwide. The aim of this study was to evaluate the clinical spectrum, prevalence, and some of the variables that are supposed to be the risk factors of this chronic disorder. Some patients have persistence of symptoms that were part of their GERD presentation, raising the concern that they are suffering from an ineffective or failing fundoplication. Unexpected postoperative heartburn, regurgitation or chest pain may result in continuation of acid-suppressive medications, or even dose escalation, without documentation. In some cases, wrap failure is implicated.

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